Targeting the unequal burden of kidney disease on black Americans

“I was in favor of that,” said Malcolm. That was Martin.

When they were tested, the brothers learned that they had those options, that the options, not the lupus, were likely to damage their kidneys. They hardly knew how to react.

“I’m still trying to fight it,” said Malcolm.

But Dr. Olabis was not surprised. Researchers believe that the only cause of kidney disease is when there is a secondary factor. The leading candidate is the body’s own antiviral response, interferon, which is abundantly produced in people with lupus.

High levels of interferon also occur in people who are not treated for HIV, as in people with Covid-19, who can be unusually catastrophic. the collapse of their kidneys if they have options. Other viral infections, including some that go unnoticed, can cause an increase in interferon, which can cause APOL1 variants. Interferon is also used as a medicine for some diseases, including cancer, and has been tested to treat patients with Covid.

So far, Malcolm and Martin can do little to control their lupus erythematosus.

Martin said he understood all that, but he was glad to know he had the options. Now he knows what he might end up with.

“I’m the person who likes to plan,” he said. “It makes a difference.”

While Dr. Olabys waits to begin his research, the pharmaceutical company Vertex has come up with its own research. However, there was no agreement on how APOL1 variants cause kidney disease, so it was not clear what drug should be blocked.

“If you do not understand the mechanism, it means that you can not measure the effects in the laboratory,” said Dr. David Altshuler, Vertex Chief Scientific Officer. “And if you can not measure the effects in the laboratory, it means you can not correct them.”

APOL1 protein was known to protect against drowsiness by puncturing disease-causing trypanosomes, causing them to swell with fluid and explode.

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